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Home › Body Atlas › Brain & Spinal Cord › Sylvian Cistern
Brain Structure Head & Skull

Sylvian Cistern

cisterna fossae lateralis cerebri

The Sylvian cistern (cistern of the lateral sulcus) is the subarachnoid space within the Sylvian fissure (lateral sulcus), containing the middle cerebral artery (M1 and M2 segments), the MCA branches, the deep Sylvian veins, and the superficial middle cerebral vein running along the opercular lip. The insular cortex (Brodmann areas 13-16) lies at the depth of the cistern. Arachnoid cysts most commonly arise in this cistern.

Anatomical Data

Structure & Connections

LocationWithin the lateral (Sylvian) fissure, between the frontal, parietal and temporal opercula and the insula
Parts
  • Cerebrospinal fluid
  • Middle cerebral artery (first and second segments) and branches
  • Superficial and deep middle cerebral veins
  • Origins of the lenticulostriate arteries
Connections
Blood SupplyMiddle cerebral artery
Biomechanics

Function & Actions

Carries the middle cerebral artery out to the surface of the hemisphere. It is a frequent site of middle cerebral artery aneurysms and arachnoid cysts.

Clinical Relevance

Clinical Notes

The Sylvian cistern is the operative field for MCA aneurysm surgery (the most common cerebral aneurysm location): transsylvian pterional craniotomy opens the Sylvian cistern to expose the MCA bifurcation aneurysm. The MCA vessels in the cistern are the primary structures at risk during temporal lobe epilepsy surgery, where the Sylvian fissure defines the lateral limit of the temporal resection. Arachnoid cysts within the Sylvian cistern produce temporal lobe compression, seizures, and headache, treated by cystoperitoneal shunting or endoscopic cyst fenestration.

Pathology

Common Injuries & Conditions

Arachnoid Cyst Expanding the Sylvian Cistern

Congenital arachnoid cyst within the Sylvian cistern progressively expands by one-way CSF flow, compressing the temporal lobe and producing headache, seizures, and developmental delay in children; MRI demonstrates the non-enhancing fluid-signal mass deforming the temporal operculum and middle fossa; endoscopic fenestration or cystoperitoneal shunting decompresses the cistern.

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